Restless Legs
Restless legs can develop because of the secondary effects of treatment or other underlying health conditions. Muscle spasms and other involuntary movements may occur if nerve cells are destroyed and the brain’s supply of dopamine is cut off. It is associated with a problem in the brain’s ability to utilize the chemical messenger dopamine, which generally helps brain cells interact and generate coordinated goal-directed muscular activity and movement. Dysfunctions in dopamine and norepinephrine metabolism in the brain have been linked to iron deficiency.
Painful lower leg cramps during activity, shortness of breath, and chest discomfort have all been linked to severe anemia. Patients with anemia often report a crawling and uncomfortable itching in their legs and feet, which often becomes more intense during sleep. Restless legs syndrome symptoms are generally more severe in the evening and throughout the night since dopamine levels typically decline towards the end of the day.
Moreover, blood and oxygen are needed to power the big leg muscles. They go into overdrive without oxygen, resulting in weariness, weakness, severe cramps, and restless leg syndrome, which causes difficulty. These symptoms are more common in persons with poor leg circulation, lung illness, or heart disease.
